The highly paid heart surgeons treat heart disease as clogged plumbing. This erroneous theory creates the largest segment of funding in hospital operating facilities. The more heart bypass operations performed the better for hospital finance. But this approach is the wrong way to keep your heart safe.
Widely Accepted Theory
The cornerstone of therapy for treatment and prevention of myocardial infarction is to remove blockages in coronary arteries that are thought to be the cause of the infarction. This, of course, is the widely accepted coronary artery thrombosis theory of infarction. According to this theory, arteries become clogged with plaque, caused by such things as smoking or high cholesterol. A clot forms a fissure in the plaque. The clot may shut off the blood flow of a coronary artery, causing a heart attack.
It is deceptively simple. The coronary arteries are clogged. No blood can flow, so the heart muscle cannot be supported and heart metabolism stops, leading to death. All of you know this by rote. It’s the party line.
But there is another theory that American medicine will never tell you. There is much clinical evidence in Germany showing remarkable success. It has been known since the 1960s.
The principle basis is that victims of fatal heart attacks have shown no evidence whatsoever of coronary occlusion (blockage). Findings can be summarized as follows:
- The coronary obstruction theory cannot adequately explain observed facts.
- The major etiologic factor underlying myocardial infarction is a primary chemical destructive process caused by unchecked metabolic acidosis (accumulation of acid) in the left ventricular tissue and substantially unrelated to coronary artery disease.
- The regular clinical use of oral g-strophanthin (a cardiac glyoside derived from the West African plant strophanthus gratus) prevents lethal myocardial tissue acidosis and, thereby, substantially reduces the incidence of myocardial infarction and completely prevents infarction deaths. Strophanthin is not available in the U.S. Drinking alkaline water on a daily basis is a good substitute to alkalize the body.
A reviewer for the publication Circulation noted: “These reports also present clear refutation of the most common explanation used to date to dismiss autopsy findings which detect NO coronary thrombi, i.e. that thrombi existed at infarction but have since lysed (dissolved) embolized or washed away.”
There appears to be no refutation in the literature of these findings. When the tissues were later dissolved in acid, the entire structure of blood vessels in the heart was revealed.
Dr. Berthold Kern, a German physician who practiced in the mid-1900s, hypothesized that bypass grafts were created naturally by the body via the collaterals when a coronary artery became blocked. Therefore, heart bypass would be redundant to a large degree.
A study by Rentrop et al. in the April 1, 1988 issue of The American Journal of Cardiology has produced results completely at odds with the coronary artery blockage theory and consistent with Kern’s hypothesis. In an accompanying editorial, Dr. Stephen Epstein of the National Heart Lung and Blood Institute summarizes Rentrop and colleagues’ “extremely important observations.”
They found that in an advanced state of the narrowing of the coronary arteries the supply of blood to the heart muscles is fully assured via collaterals that enlarge naturally in response to the blockage. Interestingly, they observed that the more the coronaries narrow, the less danger there is of heart infarction.
Kern’s second claim, i.e., his proposed “new theory” of metabolic acidosis, can be summarized as follows: Metabolic conditions in the most healthy of hearts are, at best, marginal in the constantly beating left ventricle. This is the part of the heart responsible for pumping blood to most of the body; the right ventricle merely supplying the lungs. Oxygen and energy requirements are always perilously close to available supplies, and any of several stressors may cause an oxygen/energy deficit with deterioration in oxidative metabolism and consequent development of acidosis. Lack of oxygen sets off the process of zymosis or fermentation metabolism, an anaerobic process that produces energy in the cells. This in turn lowers the pH (more acidic).
This lowering of the pH sets off a destructive chemical process, literally a suicide reaction of the cell. Lysozymal enzymes are released, causing cell self-digestion. This starts as a single point in the muscle, then many points that eventually join to form a small area of necrotic tissue. Finally, a critical mass is reached, no bigger than the head of a pin, that triggers larger and larger areas of damaged tissue leading to infarction (heart attack).
Ideally, then, the remedy to address infarction would be a restoration of pH balance to the heart muscle, thereby preventing tissue damage and fatal infarction. The problem
Kern faced was how to accomplish this without causing positive entropy (increasing the strength of muscular contraction), i.e., without putting further stress on the contracting heart muscle itself. The cardiac glycosides, including digitalis and the strophanthin byproduct known as ouabain, are known to produce such a deleterious effect and this is why they are not effective against infarction.
This is where Kern made an important rediscovery. In reviewing the literature he came across the work of Dr. Edens, who in the 1920s had reported on a qualitatively different effect of strophanthin given intravenously versus orally. Specifically, the positive enotropic effects (increasing contraction) that accompanied intravenous administration were not observed with oral administration.
This important observation has been confirmed in a study by Belz published in the European Journal of Clinical Pharmacology in 1984. Utilizing a randomized, placebo-controlled, double-blind methodology, the researchers found that the intravenous ouabain (strophanthin) produced the expected increase in cardiac entropy. However, the investigators stated quite definitely that, “…the single sublingual (oral) dose of ouabain did not exert a positive inotropic effect…”
The postulated mechanism of action, based on animal research, is that there are two receptors in the heart: “high affinity” and “low affinity.” High affinity receptors react to small concentrations of g-strophanthin, thereby avoiding positive entropy. It is thought that intravenous administration triggers low affinity receptors and thus positive entropy. Oral administration, on the other hand, triggers high affinity receptors and avoids this dangerous effect.
Kern reported results of his clinical practice in Stuttgart over the period 1947-1968 involving more than 15,000 cardiac patients. His patients treated with oral g-strophanthin experienced no fatal infarcts and only 20 non-fatal heart infarcts. These patients included many suffering infarction prior to entering the study.
In contrast with these results, government statistics for the same time period would have predicted more than 120 fatal heart infarctions and more than 400 non-fatal infarctions in this group of patients. (From WRF News, article by Wesley James entitled “G-Strophanthin: A New Approach for Heart Disease.”)
Kern observed that most myocardial infarctions (heart attacks) occur in patients without any significant obstruction of the coronary artery supplying the infarcted tissue.
Even in the U.S. in 1980, more than a dozen reports of postmortem examination of infarcted hearts have consistently failed to corroborate the coronary artery thrombosis theory of heart attack. In other words, victims of fatal heart attacks have had no evidence whatsoever of coronary occlusion.
Yet, this theory is the basis of the trillion-dollar “heart therapy” industry in the United States. This, too, is a stealth conspiracy upon heart attack victims.
An example of the degree of non-confirmation can be ascertained by the following quote from a 1980 article in Circulation: “These data support the concept that an occlusive coronary thrombus has no primary role in the pathogenesis of a myocardial infarct.”
It appears that the basis of infarction in the above article is too much acid in the system, or acidosis. Heart health is a matter of keeping normal pH balance.
We know that death is the result of progressive acid saturation. We know that the American diet has for 100 years become more and more acidic. I think that this has more and more to do with an overbalance of omega-6s coming from non-grass-fed cattle and junk food.
The American Medical Association recommends for the heart a more omega-6 diet. I believe the exact opposite. We need to reverse our omega-6 diet to an omega-3 diet by eating more fish. The Japanese have a much higher fish diet and considerably fewer heart attacks.
Fresh salmon is my favorite source of omega 3. I try to eat fish daily.
Keeping check on one’s body pH is no more complicated than keeping check on the pH of the water in a swimming pool. pH strips are available at most pharmacies. pH can be measured by saliva or urine.
Drops to make your water alkaline are available from health food stores or can be purchased online.